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Early on in the epidemic, the Chinese conducted a study that indicated that the health impact of COVID-19 may have a racial component. I mentioned the study, and immediately the usual suspects started to claim - RAMZPAUL SAYS COVID DOES NOT HURT WHITES. Of course, I said no such thing. But now we are getting further evidence that this virus may be more deadly based on your genetics.

ramzpaul 8 June 12
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There are going to be many aspects to this most likely. For example, if we determine there is a strong correlation between Type O blood and lower levels of ACE 2 receptor expression in the lungs and upper respiratory system, with ACE 2 being strongly suggested as the point of binding and entry for the virus, what would that tell us? Correlation and causation are difficult to delineate often, for sure. For example, those with alleles for lowered ace2 expression may "just so happen" to have alleles for ABO that lack enzymatic activity- resulting in the O phenotype. However, that of course assumes that ACE 2 expression is independent of pathways involving ABO determination, and I am unaware of what interplay there might be between the two if any. If there is mechanistic interplay, ace2 expression may be dependent to some degree on ABO genotype in combination with the alleles for the ace2 gene. If there is no overlap or involvement of ABO determination in the expression of ACE 2 at the cell's surface, it may simply point toward population allelic frequencies as the underlying correlation. This is because the genes for each are located on separate chromosomes and surely not involved in some kind of linkage disequilibrium due to infrequent recombination. In that model, you would expect to see a correlation between allelic frequencies that result in lowered ACE 2 expression and those of the O ABO alleles in populations of people where O Type seems to have been correlated with lowered incidence of infection. Regional homogeny in China could explain this, as you may have had populations of people living in certain places for long periods of time and having inherited both alleles on their separate chromosomes from some ancestral population, resulting in increased inheritance of both. There are many other factors as well, and ultimately we would need to see the results from various populations of people in relation to the Type O observation and not necessarily focus on the China observation- again because if ace2 expression is not dependent on blood type pathways at all it would surely reveal that result in populations of people with higher ACE 2 levels even in combination with the O blood type. In that scenario do we see increased "shielding" as a result, etc? Many questions to ask in order to understand the nature of those that seem to be more resistant.

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@ramzpaul I'm surprised that more diseases haven't been flagged as having different effects on different races. Some differences are known. For example, diabetes seems to affect South Asians worse than others. Here's a link: [[ncbi.nlm.nih.gov]]

From the abstract:

"...they [south Asians] experience significant morbidity and mortality from complications of diabetes, most notably coronary artery disease, cerebrovascular disease and chronic kidney disease."

The medical community cannot afford to live in the Orwellian fantasy world of today's leftists where the practices of doublethink and crimestop prevail. But of course sometimes they do.

Choppy Level 5 June 12, 2020

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